I understood that ethanol increases Golgi cell firing by inhibiting the Na+/K+ ATPase channels, which makes granule cells receive more GABAergic input from the GABA that Golgi cells relase. Some paragraph in this paper are not clear to me. It says that

ethanol increased spontaneous firing frequency and depolarized the membrane potential in the perforated-patch configuration, but also that ethanol does not significantly affect firing frequency or membrane potential response to current injection.

How does it work?


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